Back

The aging lung mucosa: A proteomics study

Garcia-Vilanova, A.; Olmo-Fontanez, A. M.; Moliva, J. I.; Allue Guardia, A.; Singh, H.; Merrit, R. E.; Caceres, D. M.; Peters, J.; Wang, Y.; Schlesinger, L. S.; Turner, J.; Weintraub, S. T.; Torrelles, J. B.

2021-09-20 systems biology
10.1101/2021.09.14.460375 bioRxiv
Show abstract

The elderly population is at increased risk of acute and chronic respiratory infections and other pulmonary diseases, and it is estimated that this population will double in the next 30 years. Biochemical changes in the lung alveolar mucosa and lung cells alter local immune response as we age, creating opportunities for invading pathogens to establish successful infections. Indeed, the lungs of the elderly are a pro-inflammatory, pro-oxidative, dysregulated environment but this environment has remained understudied. We performed a comprehensive, quantitative proteomic profile of the lung mucosa in the elderly, developing insight into the molecular fingerprints, pathways, and regulatory networks that characterize the lung in old age. We identified neutrophils in the lungs of elderly individuals as possible contributors to dysregulated lung tissue environment. This study establishes a baseline for future investigations to develop strategies to mitigate susceptibility to respiratory infections in the elderly.

Matching journals

The top 9 journals account for 50% of the predicted probability mass.

50% of probability mass above

"Similar papers" are the closest papers from that journal in the model's embedding space. They show what the match is built on, but the ranking comes mostly from a classifier over the whole training set, not from these examples alone.