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An adult clock component links circadian rhythms to pancreatic β-cell maturation

Montalvo, A. P.; Gruskin, Z. L.; Leduc, A.; Liu, M.; Gao, Z.; Ahn, J. H.; Straubhaar, J. R.; Slavov, N.; Alvarez-Dominguez, J. R.

2023-08-12 cell biology
10.1101/2023.08.11.552890 bioRxiv
Show abstract

Circadian clocks attune metabolism to daily energy cycles, but how they regulate functional maturation of metabolic tissues is poorly understood. Here we show that DEC1, a clock transcription factor induced in adult islet {beta} cells, coordinates their glucose responsiveness by synchronizing energetic and secretory rhythms. DEC1 binds and regulates maturity-linked genes to integrate insulin exocytosis with {beta}-cell energy metabolism, and {beta}-cell-specific Dec1 ablation disrupts their transcription synchrony. Dec1-disrupted mice develop lifelong glucose intolerance and insulin deficiency, despite normal islet formation. Metabolic dysfunction upon {beta}-cell Dec1 loss stems from poor coupling of islet insulin secretion to glucose metabolism, reminiscent of fetal/neonatal islet immaturity. We link stunted maturation to a deficit in circadian bioenergetics, prompted by compromised glucose utilization, mitochondrial dynamics, and respiratory metabolism, which is rescued by increased metabolic flux. Thus, DEC1 links circadian clockwork to {beta}-cell metabolic maturation, revealing a hierarchy for how the clock programs metabolic tissue specialization.

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