Back

Somatic hypermutation spectra are independent of the local transcriptional and epigenetic landscape

Schoeberl, U. E.; Fitz, J.; Froussios, K.; Valieris, R.; Makharova, M.; Ourailidis, I.; Bauer, B.; Neumann, T.; Wiedemann, E.-M.; Steininger, M.; Garcia, A. C.; Mastrovito, M.; Mouquet, H.; Silva, I.; Pavri, R.

2022-05-21 immunology
10.1101/2022.05.21.492925 bioRxiv
Show abstract

Somatic hypermutation (SHM) of immunoglobulin variable (V) regions modulates antibody-antigen affinity is initiated by activation-induced cytidine deaminase (AID) on single-stranded DNA (ssDNA). Transcription is essential for SHM and AID target genes harbor activating chromatin marks and RNA polymerase II (Pol II) stalling, leading to the model that these features favor higher rates of mutagenesis. However, whether such relationships exist within V regions is undetermined. Here, we directly compared SHM and nascent transcription across four V regions and 275 non-immunoglobulin SHM targets at single-nucleotide resolution using precision run-on sequencing (PRO-seq). Although locales of Pol II enrichment and zones of Pol II stalling were detected within V regions, their correlation with SHM was not statistically significant. Moreover, SHM was robust against major reductions of activating epigenetic marks and transcription. This data suggests that SHM patterns and spectra are established independently of specific local nascent transcriptional features.

Matching journals

The top 4 journals account for 50% of the predicted probability mass.

50% of probability mass above

"Similar papers" are the closest papers from that journal in the model's embedding space. They show what the match is built on, but the ranking comes mostly from a classifier over the whole training set, not from these examples alone.